Reading list
The articles behind the avoidance task, the objects, the molecular measurements and the human anxiety findings.
Memory and brain measurements
The memory-model articles sit beside the Semax protein and gene-expression studies. Selank’s rat findings add another set of molecular observations. The memory experiments and brain-signal notes discuss the particular outcomes that interested me.
Anxiety, immunity and an interaction
The human Selank reports concern anxiety and immune measurements. The Semax monoamine article concerns mice. Those differences recur in the human findings, where the appeal of a helpful companion meets some less comfortable questions.
- Neuroprotective and antiamnesic effects of Semax during experimental ischemic infarction of the cerebral cortex ↗
- The Potential of the Peptide Drug Semax and Its Derivative for Correcting Pathological Impairments in the Animal Model of Alzheimer's Disease ↗
- Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain ↗
- Neurotrophin gene expression in rat brain under the action of Semax, an analogue of ACTH(4-10) ↗
- Intranasal administration of the peptide Selank regulates BDNF expression in the rat hippocampus in vivo ↗
- Selank Administration Affects the Expression of Some Genes Involved in GABAergic Neurotransmission ↗
- Efficacy and possible mechanisms of action of a new peptide anxiolytic medication (selank) in the therapy of generalized anxiety disorders and neurasthenia ↗
- Immunomodulatory effects of selank in patients with anxiety-asthenic disorders ↗
- Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents ↗